@article{oai:kumadai.repo.nii.ac.jp:00021862,
 author = {白川, 久志 and Shirakawa, Hisashi and 香月, 博志 and 久米, 利明 and Kume, Toshiaki and 金子, 周司 and Kaneko, Shuji and 赤池, 昭紀 and Akaike, Akinori and 白川, 久志 and Shirakawa, Hisashi and 香月, 博志 and Katsuki, Hiroshi and 久米, 利明 and Kume, Toshiaki and 金子, 周司 and Kaneko, Shuji and 赤池, 昭紀 and Akaike, Akinori},
 issue = {7},
 journal = {British Journal of Pharmacology},
 month = {Apr},
 note = {application/pdf, 論文(Article), Aminoglutethimide is a clinically available drug that suppresses steroid biosynthesis by inhibiting enzymes such as cytochrome P450scc and aromatase. Because several members of neurosteroids regulate glutamate receptors, we investigated the effect of aminoglutethimide on cell death induced by overactivation of glutamate receptors in CNS neurons., http://www.nature.com/bjp/index.html},
 pages = {729--736},
 title = {Aminoglutethimide prevents excitotoxic and ischemic injuries in cortical neurons},
 volume = {147},
 year = {2006},
 yomi = {シラカワ, ヒサシ and クメ, トシアキ and カネコ, シュウジ and アカイケ, アキノリ and シラカワ, ヒサシ and カツキ, ヒロシ and クメ, トシアキ and カネコ, シュウジ and アカイケ, アキノリ}
}